By Irma Pereira MT (ASCP) SH, Tracy I. George MD, Daniel A. Arber MD
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Extra info for Atlas of Peripheral Blood: The Primary Diagnostic Tool
Color Atlas of Hematology. An Illustrated Field Guide Based on Proficiency Testing. Northfield, IL: College of American Pathologists; 1998. Perkins SL. Examination of the blood and bone marrow. , eds. Wintrobe’s Clinical Hematology. 12th ed. ; 2008. Pierre RV. Red cell morphology and the peripheral blood film. Clin Lab Med. 2002;22(1):25–61. 3 Anemia Due to Abnormal or Impaired Hemoglobin Synthesis Anemias due to abnormal hemoglobin synthesis or impaired hemoglobin synthesis are characterized by a microcytic anemia.
Splenomegaly or hypersplenism is another factor that can induce mechanical destruction of RBCs leading to normocytic normochromic anemia. Leukocytes and platelets may also decrease in number secondary to sequestration, resulting in additional cytopenias. INTRACORPUSCULAR Oxidative Hemolytic Anemia Glucose-6-phosphate dehydrogenase (G6PD) and pyruvate kinase (PK) deficiencies are the two most common hereditary RBC enzyme disorders that can cause hemolytic anemia (Fig. 6). G6PD deficiency: This is the most common RBC enzyme deficiency affecting more than 400 million people.
INTRACORPUSCULAR Oxidative Hemolytic Anemia Glucose-6-phosphate dehydrogenase (G6PD) and pyruvate kinase (PK) deficiencies are the two most common hereditary RBC enzyme disorders that can cause hemolytic anemia (Fig. 6). G6PD deficiency: This is the most common RBC enzyme deficiency affecting more than 400 million people. The incidence is highest in Africans, people of Mediterranean descent, and Southeast Asians. 2 A: A remarkable number of schistocytes are present in this patient with microangiopathic hemolytic anemia due to hemolytic uremic syndrome.